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wild-type and p75 ntr knockout mice  (Jackson Laboratory)

 
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    Structured Review

    Jackson Laboratory wild-type and p75 ntr knockout mice
    A summary of sources of antibodies used to detect protein expression using Western Blot.
    Wild Type And P75 Ntr Knockout Mice, supplied by Jackson Laboratory, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/wild-type and p75 ntr knockout mice/product/Jackson Laboratory
    Average 90 stars, based on 1 article reviews
    wild-type and p75 ntr knockout mice - by Bioz Stars, 2026-03
    90/100 stars

    Images

    1) Product Images from "Deletion of the Neurotrophin Receptor p75 NTR Prevents Diabetes-Induced Retinal Acellular Capillaries in Streptozotocin-Induced Mouse Diabetic Model"

    Article Title: Deletion of the Neurotrophin Receptor p75 NTR Prevents Diabetes-Induced Retinal Acellular Capillaries in Streptozotocin-Induced Mouse Diabetic Model

    Journal: Journal of diabetes, metabolic disorders & control

    doi: 10.15406/jdmdc.2017.04.00129

    A summary of sources of antibodies used to detect protein expression using Western Blot.
    Figure Legend Snippet: A summary of sources of antibodies used to detect protein expression using Western Blot.

    Techniques Used: Expressing, Western Blot

    Genetic deletion of p75NTR mitigated diabetes-induced acellular capillaries. (C–F): Representative images of trypsinized-retina from WT or p75NTR KO controls or diabetic mice. (G): Quantitative data showing significant increase in retinal acellular capillaries after 6-month of diabetes in WT-diabetic but not in p75 KO-diabetic animals compared to respective controls. (A–B): Representative images of a typical occluded (acellular) capillary in a trypsinized retina of diabetic animals double-labelled with anti-Collagen IV (Green) and CD31 (Red).
    Figure Legend Snippet: Genetic deletion of p75NTR mitigated diabetes-induced acellular capillaries. (C–F): Representative images of trypsinized-retina from WT or p75NTR KO controls or diabetic mice. (G): Quantitative data showing significant increase in retinal acellular capillaries after 6-month of diabetes in WT-diabetic but not in p75 KO-diabetic animals compared to respective controls. (A–B): Representative images of a typical occluded (acellular) capillary in a trypsinized retina of diabetic animals double-labelled with anti-Collagen IV (Green) and CD31 (Red).

    Techniques Used:



    Similar Products

    wild-type and p75 ntr knockout mice  (Jackson Laboratory)
    90
    Jackson Laboratory wild-type and p75 ntr knockout mice
    A summary of sources of antibodies used to detect protein expression using Western Blot.
    Wild Type And P75 Ntr Knockout Mice, supplied by Jackson Laboratory, used in various techniques. Bioz Stars score: 90/100, based on 1 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/result/wild-type and p75 ntr knockout mice/product/Jackson Laboratory
    Average 90 stars, based on 1 article reviews
    wild-type and p75 ntr knockout mice - by Bioz Stars, 2026-03
    90/100 stars
    		  Buy from Supplier

    Image Search Results


    A summary of sources of antibodies used to detect protein expression using Western Blot.

    Journal: Journal of diabetes, metabolic disorders & control

    Article Title: Deletion of the Neurotrophin Receptor p75 NTR Prevents Diabetes-Induced Retinal Acellular Capillaries in Streptozotocin-Induced Mouse Diabetic Model

    doi: 10.15406/jdmdc.2017.04.00129

    Figure Lengend Snippet: A summary of sources of antibodies used to detect protein expression using Western Blot.

    Article Snippet: Induction of diabetes Male 8-week old littermates of wild-type and p75 NTR knockout mice (~25g) from our colony with founders originally purchased from Jackson laboratory (Bar Harbor, ME, USA).

    Techniques: Expressing, Western Blot

    Genetic deletion of p75NTR mitigated diabetes-induced acellular capillaries. (C–F): Representative images of trypsinized-retina from WT or p75NTR KO controls or diabetic mice. (G): Quantitative data showing significant increase in retinal acellular capillaries after 6-month of diabetes in WT-diabetic but not in p75 KO-diabetic animals compared to respective controls. (A–B): Representative images of a typical occluded (acellular) capillary in a trypsinized retina of diabetic animals double-labelled with anti-Collagen IV (Green) and CD31 (Red).

    Journal: Journal of diabetes, metabolic disorders & control

    Article Title: Deletion of the Neurotrophin Receptor p75 NTR Prevents Diabetes-Induced Retinal Acellular Capillaries in Streptozotocin-Induced Mouse Diabetic Model

    doi: 10.15406/jdmdc.2017.04.00129

    Figure Lengend Snippet: Genetic deletion of p75NTR mitigated diabetes-induced acellular capillaries. (C–F): Representative images of trypsinized-retina from WT or p75NTR KO controls or diabetic mice. (G): Quantitative data showing significant increase in retinal acellular capillaries after 6-month of diabetes in WT-diabetic but not in p75 KO-diabetic animals compared to respective controls. (A–B): Representative images of a typical occluded (acellular) capillary in a trypsinized retina of diabetic animals double-labelled with anti-Collagen IV (Green) and CD31 (Red).

    Article Snippet: Induction of diabetes Male 8-week old littermates of wild-type and p75 NTR knockout mice (~25g) from our colony with founders originally purchased from Jackson laboratory (Bar Harbor, ME, USA).

    Techniques: